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Artigo de periodico
Urate as a physiological substrate for myeloperoxidase: implications for hyperuricemia and inflammation (2011)
Meotti, Flavia Carla ; Jameson, Guy N. L; Turner, Rufus; Harwood, D. Tim; Stockwell, Samantha; Rees, Martin D; Thomas, Shane R; Kettle, Anthony J
Fonte: Journal of Biological Chemistry. Unidade: IQ
Assuntos: INFLAMAÇÃO, BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MEOTTI, Flavia Carla et al. Urate as a physiological substrate for myeloperoxidase: implications for hyperuricemia and inflammation. Journal of Biological Chemistry, v. 286, n. 15, p. 12901-12911, 2011Tradução . . Disponível em: https://doi.org/10.1074/jbc.m110.172460. Acesso em: 17 maio 2024.
APA
Meotti, F. C., Jameson, G. N. L., Turner, R., Harwood, D. T., Stockwell, S., Rees, M. D., et al. (2011). Urate as a physiological substrate for myeloperoxidase: implications for hyperuricemia and inflammation. Journal of Biological Chemistry, 286( 15), 12901-12911. doi:10.1074/jbc.m110.172460
NLM
Meotti FC, Jameson GNL, Turner R, Harwood DT, Stockwell S, Rees MD, Thomas SR, Kettle AJ. Urate as a physiological substrate for myeloperoxidase: implications for hyperuricemia and inflammation [Internet]. Journal of Biological Chemistry. 2011 ; 286( 15): 12901-12911.[citado 2024 maio 17 ] Available from: https://doi.org/10.1074/jbc.m110.172460
Vancouver
Meotti FC, Jameson GNL, Turner R, Harwood DT, Stockwell S, Rees MD, Thomas SR, Kettle AJ. Urate as a physiological substrate for myeloperoxidase: implications for hyperuricemia and inflammation [Internet]. Journal of Biological Chemistry. 2011 ; 286( 15): 12901-12911.[citado 2024 maio 17 ] Available from: https://doi.org/10.1074/jbc.m110.172460
Artigo de periodico
Activation of cannabinoid receptors by the pentacyclic triterpene 'alpha','beta'-amyrin inhibits inflammatory and neuropathic persistent pain in mice (2011)
Silva, Kathryn Ana Bortolim Simão da; Paszcuk, Ana F; Passos, Giselle F; Silva, Eduardo Santos da; Bento, Allison Freire; Meotti, Flavia Carla ; Calixto, João Baptista
Fonte: PAIN. Unidade: IQ
Assuntos: CANNABIDACEAE, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Kathryn Ana Bortolim Simão da et al. Activation of cannabinoid receptors by the pentacyclic triterpene 'alpha','beta'-amyrin inhibits inflammatory and neuropathic persistent pain in mice. PAIN, v. 152, n. 8, p. 1872-1887, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.pain.2011.04.005. Acesso em: 17 maio 2024.
APA
Silva, K. A. B. S. da, Paszcuk, A. F., Passos, G. F., Silva, E. S. da, Bento, A. F., Meotti, F. C., & Calixto, J. B. (2011). Activation of cannabinoid receptors by the pentacyclic triterpene 'alpha','beta'-amyrin inhibits inflammatory and neuropathic persistent pain in mice. PAIN, 152( 8), 1872-1887. doi:10.1016/j.pain.2011.04.005
NLM
Silva KABS da, Paszcuk AF, Passos GF, Silva ES da, Bento AF, Meotti FC, Calixto JB. Activation of cannabinoid receptors by the pentacyclic triterpene 'alpha','beta'-amyrin inhibits inflammatory and neuropathic persistent pain in mice [Internet]. PAIN. 2011 ; 152( 8): 1872-1887.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.pain.2011.04.005
Vancouver
Silva KABS da, Paszcuk AF, Passos GF, Silva ES da, Bento AF, Meotti FC, Calixto JB. Activation of cannabinoid receptors by the pentacyclic triterpene 'alpha','beta'-amyrin inhibits inflammatory and neuropathic persistent pain in mice [Internet]. PAIN. 2011 ; 152( 8): 1872-1887.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.pain.2011.04.005
Artigo de periodico
The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice (2011)
Bicca, M. A; Figueiredo, C. P; Piermartiri, T. C; Meotti, Flavia Carla ; Bouzon, Zenilda Laurita; Tasca, Carla I; Medeiros, R; Calixto, João Baptista
Fonte: Neuroscience. Unidade: IQ
Assuntos: RECEPTORES, GLUTAMATOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BICCA, M. A et al. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice. Neuroscience, v. 192, n. 29, p. 631-641, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2011.06.038. Acesso em: 17 maio 2024.
APA
Bicca, M. A., Figueiredo, C. P., Piermartiri, T. C., Meotti, F. C., Bouzon, Z. L., Tasca, C. I., et al. (2011). The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice. Neuroscience, 192( 29), 631-641. doi:10.1016/j.neuroscience.2011.06.038
NLM
Bicca MA, Figueiredo CP, Piermartiri TC, Meotti FC, Bouzon ZL, Tasca CI, Medeiros R, Calixto JB. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice [Internet]. Neuroscience. 2011 ; 192( 29): 631-641.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.06.038
Vancouver
Bicca MA, Figueiredo CP, Piermartiri TC, Meotti FC, Bouzon ZL, Tasca CI, Medeiros R, Calixto JB. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice [Internet]. Neuroscience. 2011 ; 192( 29): 631-641.[citado 2024 maio 17 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.06.038

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